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Overexpression of the human DEK oncogene reprograms cellular metabolism and promotes glycolysis
Significance of DEK overexpression for the prognostic evaluation of non-small cell lung carcinoma
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Immiscible inclusion bodies formed by polyglutamine and poly(glycine-alanine) are enriched with distinct proteomes but converge in proteins that are risk factors for disease and involved in protein degradation
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DEK-CAN molecular monitoring of myeloid malignancies could aid therapeutic stratification | Leukemia
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Overexpression of the human DEK oncogene reprograms cellular metabolism and promotes glycolysis
Significance of DEK overexpression for the prognostic evaluation of non-small cell lung carcinoma
DEK-CAN molecular monitoring of myeloid malignancies could aid therapeutic stratification | Leukemia
Epithelial‑mesenchymal transition in colorectal carcinoma cells is mediated by DEK/IMP3
Overexpression of the human DEK oncogene reprograms cellular metabolism and promotes glycolysis
Monosomal karyotype as an adverse prognostic factor in patients with acute myeloid leukemia treated with allogeneic hematopoietic stem-cell transplantation in first complete remission: a retrospective survey on behalf of the ALWP of
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NUP98-fusion transcripts characterize different biological entities within acute myeloid leukemia: a report from the AIEOP-AML group | Leukemia
Epithelial‑mesenchymal transition in colorectal carcinoma cells is mediated by DEK/IMP3
Significance of DEK overexpression for the prognostic evaluation of non-small cell lung carcinoma
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